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PEPID
Subsections
Impetigo

Dermatology | Infectious Diseases

Impetigo

Background

  1. Definition
    • Infection of the superficial layers of the epidermis that is highly contagious and most common due to group A Streptococcus (GAS), Staphylococcus aureus, or both
  2. Synopsis
    • Recently, MRSA is an important etiology to rule out
    • Most commonly presents as erythematous plaques w/ a yellow crust
      • May be itchy or painful
    • Highly contagious, spreads easily
    • Diagnosis
      • Symptoms and clinical manifestations
    • Treatment
      • Topical and oral antibiotics
      • Symptom control

Pathophysiology

  1. Etiology/Risk Factors
    • Most common in children 2-5 yo
    • Triggers breaking down skin and increasing susceptibility
      • Burns
      • Herpes
      • Insect bites
      • Lice
      • Scabies
      • Scratching
      • Trauma
      • Varicella
    • Malnutrition
    • Immunosuppression
    • Daycare attendance
    • Overcrowding
    • Diabetes
    • Poor hygiene
  2. Pathology
    • Superficial skin infection
    • Usual colonization in nares/pharynx
      • Non-bullous: most common
        • Start as papules that evolve to vesicles with erythematous bases
        • Then to pustules that rupture to form golden “honey-colored” crust
      • Bullous: usually younger children
        • Vesicles enlarge to form flaccid bullae
        • Later rupture and leave a brown-colored crust
      • Strains of S. aureus with exfoliative toxins
        • Loss of cell adhesion in superficial epidermis
      • Ecthyma: ulcerative impetigo extending into deep dermis
        • “Punched-out” ulcers with yellow crust and violaceous edges
    • Contagious, transferred through fomites (clothes, toys)
      • Handwashing
        • Essential to prevent spread
      • May be acquired at birth if PROM
  3. Epidemiology
    • Incidence/Prevalence
      • 10% of all skin disorders affecting children
      • Most common in
        • Warm, humid areas
        • Southeast US
      • Peak incidence
        • Age 2-5 years old
      • Higher incidence
        • Areas with high crowding, poor hygiene
    • Morbidity/Mortality
      • Can progress to any Staph/Strep disease
        • Cellulitis
        • Cutaneous abscess (esp. MRSA) lymphadenitis
        • Staph scalded skin syndrome
        • Meningitis
        • Sepsis
      • Post-streptococcal glomerulonephritis
        • Antibiotic tx does not reduce risk
      • Neonates
        • More susceptible to developing meningitis/sepsis

Diagnostics

  1. History/Symptoms
    • Hx of skin trauma
      • Bites, scratches
    • Rash on exposed
      • Skin of face (mouth/nose)
      • Extremities
      • Begins painless, occasionally pruritic
      • Yellow/honey-colored lesions (crusted)
    • Bullous lesions that spontaneously rupture may be present
      • May have associated F/D, weakness
    • Hx of acute glomerulonephritis (Strep)
  2. Physical Exam/Signs
    • Nonbullous impetigo
      • 1-3 mm macule evolves into
      • Serous discharge crusted plaques with erythematous base
      • Weeping, erythematous, shallow erosions or ulcerations
      • Heals without scarring
      • +/- regional LAD
    • Bullous impetigo
      • Fragile, fluid-filled vesicle or bulla < 3 cm (View image)
      • Most commonly
        • Face
        • Buttocks
        • Trunk
        • Perineum
        • Extremities
      • Ruptures in 1-2 days
        • Resulting in superficial erosion with moist, erythematous base
      • May have scalded skin appearance
      • Usually LAD
    • Ecthyma
      • Transient vesicular lesions
        • With thick, adherent crust
      • Central area
        • Becomes necrotic
      • Erythematous, indurated periphery
        • Occasionally purulent
      • Central ulcer scarring
    • Edema, HTN
      • Glomerulonephritis
  3. Labs/Tests
    • Clinical diagnosis
    • Urinalysis
      • Rule-out glomerulonephritis
    • Culture & Stain for unknown Dx
      • Nare cultures
        • Potential carriers, close contacts
      • C&S
        • Treatment failures for Abx adjustment
    • Bacterial cultures
      • Obtain if MRSA suspected
    • ASO
      • Not indicated
      • Response is weak from impetigo alone
  4. Imaging
    • Usually not routine
      • Unless indicated by underlying pathology
        • Deep wound
        • Organ failure signs
  5. Differential Diagnosis

Treatment

  1. Initial/Prep/Goals
    • Contact isolation
    • Local wound care
      • Wet dressings
      • Cleansing of lesions
    • Insufficient evidence
      • To compare effectiveness of topical vs oral Abx
        • In widespread impetigo
  2. Medical/Pharmaceutical
  3. Complications
    • Post-streptococcal glomerulonephritis
      • May occur 2-3 weeks after skin infection
    • Rheumatic fever
    • Meningitis
      • May develop in neonates
    • Renal failure
    • Septic arthritis
    • Scarlet fever
    • Sepsis
    • Staphylococcal scaled skin syndrome
  4. Prevention
    • Wash body and hair often with soap and clean, running water
    • Wash the clothes, linens, and towels of anyone with impetigo every day
    • Don't share clothes, linens, or towels with anyone who has impetigo

Disposition

  1. Admission Criteria
    • Admit if severe secondary complications
  2. Consult(s)
    • Neonatology for neonate infections
  3. Discharge/Follow-Up instructions
    • Isolation from school until > 24 hours after Abx treatment
      • Patient no longer infectious
    • Follow up with PCP as appropriate
      • Follow-up within 7-10 days; lesions should be healed
        • If still present: C&S lesions, add PO Abx
      • Examine, treat family members, close contacts
        • Mupirocin cream to nares in carriers (monthly if chronic)
      • Monitor for glomerulonephritis:
        • May occur up to 5 weeks after disease

References

  1. Habif TP. Bacterial infections. Clinical Dermatology. 5th ed. St. Louis, Mo: Mosby-Elsevier, 2009;Chapter 9
  2. Mandell GL, Bennett JE, Dolin R, Pasternack MS, Swartz MN, et al. Cellulitis, necrotizing fasciitis, and subcutaneous tissue infections. Principles and Practice of Infectious Diseases, 7th ed. Philadelphia, Pa: Elsevier-Churchill Livingstone, 2009;Chapter 90
  3. Moulin F, Quinet B, Raymond J, Gillet Y, Cohen R. [Managing children skin and soft tissue infections]. Arch Pediatr. Oct 2008;15(Suppl 2):S62-67
  4. Bernard P. Management of common bacterial infections of the skin. Curr Opin Infect Dis. Apr 2008;21(2):pp.122-128
  5. Cole C, Gazewood J. Diagnosis and treatment of impetigo. Am Fam Physician, Mar 15, 2007;75(6):pp.859-864
  6. Mertz PM, Marshall DA, Eaglstein WH, Piovanetti Y, Montalvo J. Topical mupirocin treatment of impetigo is equal to oral erythromycin therapy. Arch Dermatol. Aug 1989;125(8):pp.1069-1073
  7. Koning S, van der Sande R, Verhagen AP, et al. Interventions for impetigo. Cochrane Database Syst Rev. Jan 18, 2012.
  8. Cydulka RK, Stewart MH Dermatologic Presentations: Impetigo. In Marx JA, Hockberger RS, Walls RM eds. Rosen’s Emergency Medicine Concepts and Clinical Practice. 5th ed. St. Louis, MO: Mosby; 2002: 1640
  9. Malley R. Skin and Soft Tissue Infections: Impetigo. In Tintinalli JE, Kelen GD, Stapczynski JS eds. Emergency Medicine: A Comprehensive Study Guide. 6th ed. New York, NY: McGraw-Hill; 2004:779-780
  10. McMillan JA, Feigin RD, DeAngelis C, Jones MD. Oski's Pediatrics: Principles & Practice, 4th ed, Philadelphia, PA:Lippincott Williams & Wilkins, 2006;Chapter 74
  11. Cole C, Gazewood J. Diagnosis and treatment of impetigo. Am Fam Physician. 2007 Mar 15. 75(6):859-64.
  12. Impetigo. StatPearls [Internet]. Available at: https://www.ncbi.nlm.nih.gov/books/NBK430974/. [Accessed September 2024]
  13. Centers for Disease Control and Prevention (CDC). About Impetigo. Available at: https://www.cdc.gov/group-a-strep/about/impetigo.html. [Accessed September 2024]
  14. Centers for Disease Control and Prevention (CDC). Preventing Group A Strep Infection. Available at: https://www.cdc.gov/group-a-strep/prevention/index.html. [Accessed September 2024]

Contributor(s)

  1. Hilbert, Sue-Lin, MD
  2. Ballarin, Daniel, MD
  3. Singh, Ajaydeep, MD

Updated/Reviewed: September 2024